Weight Loss Naturopath & Nutritionist · Australia Wide
You've been told it's just your age, just your hormones, just willpower, or you're doing everything right and the weight will not shift.
Either way, your weight, your energy, your appetite, and your metabolism are not where they should be.
They're not telling you everything.
You've done everything you used to do, and the weight is not coming off.
The workouts that used to work don't. The 1200-calorie phase, the 16:8, the 30-day challenge, none of it has shifted what's around your middle.
You're hungry an hour after a meal you would have called filling two years ago.
You're tired in a way that no amount of sleep fixes, and your jeans are tight around the belly even though everything below the waist still fits the way it used to.
By 3pm you're wiped out, standing in the pantry eating things you said you wouldn't.
And every time you ask a doctor, the answer is some version of "well, you're getting older" or "have you tried eating less and moving more."
This page is for women who want to understand what is going on with their body & answers.
You're between 25 and 60, and something has shifted, slowly enough that you didn't catch it at first.
This is what I see in clinic every week.
You have noticed the weight creep on over the past few years and you cannot trace it to anything specific (no major life change, no big shift in eating, no new medication).
Your weight sits around your belly and your waist now in a way it never used to, and your arms and your bra line are heavier than they were 2 years ago.
Your jeans are tight around the middle but still fit your hips and thighs.
You are tired in a way that no amount of sleep fixes, particularly between 2 and 4pm.
You are hungry within an hour or two of meals that should be filling, and you are reaching for sugar or carbohydrates by mid-afternoon.
You wake between 2 and 4am with your mind running, and you cannot get back to sleep for an hour or more.
Your cycle is changing (heavier, lighter, shorter, longer, or more unpredictable) and the weight gain is happening alongside it.
You are bloated by the end of the day, and your fingers are puffy in a way they were not 18 months ago.
You have tried 1200 calories, the 16:8, the cleanse, the strength phase, the cardio phase, the 30-day reset, and none of it has held.
You have been told by a GP that your bloods are fine, your thyroid is fine, your hormones are fine.
You have been told to eat less and move more, and you are already doing both.
You are on, considering, or recently off a GLP-1 medication (mounjaro, ozempic, wegovy) and you want to do the foundational work alongside it or after it.
You can feel your body has changed and you cannot work out what is going on, because the things that used to work are not working anymore.
You are exhausted by the conversation about your weight and you want a clinical answer instead of another diet.
Stubborn weight gain in women is rarely a calorie problem. It is a hormonal, metabolic, and inflammatory problem. When the same way of eating that used to work stops working, when the weight creeps on around the middle without a clear cause, when the scale will not move regardless of how clean the food is, the issue is almost always sitting underneath the calorie equation, in the systems that decide whether your body stores fat or burns it.
The engine running underneath most of it is insulin resistance. Insulin is the hormone your pancreas releases every time you eat to move glucose out of your bloodstream and into your cells for energy or storage. When your cells have been exposed to high insulin for years (from frequent carbohydrate-heavy meals, chronic stress, poor sleep, or all three running at once), they become less responsive to its signal. Your pancreas produces more insulin to compensate, and the higher your circulating insulin sits, the more your body is locked into fat storage mode. While insulin is elevated, fat cells cannot release stored fat for energy, which is why you can be eating well and exercising and still seeing nothing on the scale. Insulin resistance is far more common in Australian women than the standard pathology suggests, and it is a precursor to type 2 diabetes (more on this from Diabetes Australia).
Cortisol sits alongside insulin in the body composition picture. When stress is chronic (which for most women in their 30s, 40s, and 50s means work pressure, caregiving, financial load, sleep loss, and perimenopausal hormonal fluctuations stacked on top of each other), cortisol stays elevated. Cortisol promotes fat storage specifically around the abdomen and the organs (visceral fat), because abdominal fat cells have approximately four times more cortisol receptors than fat cells anywhere else in the body. Cortisol also raises blood sugar by signalling the liver to release stored glucose, which raises insulin, which loops back into the storage pattern. The midsection weight that will not budge, the thicker waist, the jeans that fit at the hips but pinch at the belly, are usually a cortisol-insulin presentation, and the standard advice of eating less and moving more does not address it.
Hormones add a third layer, particularly for women in perimenopause and menopause. As oestrogen levels fluctuate and decline, insulin sensitivity drops. Progesterone, the calming hormone, falls earlier than oestrogen and shifts how the body handles fluid retention, sleep, mood, and the way fat is distributed. Thyroid function gets pulled into this as well, because elevated insulin and cortisol both impair the conversion of T4 (the inactive thyroid hormone your gland produces) into T3 (the active form your cells need), which slows your resting metabolic rate. The result is a body that stores more easily, burns less efficiently, and responds to dietary change differently than it did 5 or 10 years ago.
Your gut, your liver, and your muscle mass complete the picture. The gut microbiome influences how much glucose your body extracts from food, how efficiently used hormones are cleared, and how much inflammation is circulating, all of which affect weight. The liver clears excess oestrogen, processes used cortisol, and converts T4 to T3, and a congested liver slows every one of those functions. Muscle mass is the engine of your metabolism, and women who have spent years under-eating or doing only cardio often lose the very tissue that determines how many calories they burn at rest. When muscle drops, metabolic rate drops with it, and the body becomes more efficient at storing what comes in.
I find in my clinic that women presenting with stubborn weight gain almost always have several of these systems involved at once, which is why a single intervention (a new diet, a new exercise plan, a new supplement) rarely shifts the picture. The work is identifying which systems are out of balance and supporting them in the right order.
These are the symptoms that come up in my clinic every week.
If you are ticking multiple, it is worth investigating rather than waiting to see if it gets worse.
Weight gain around the belly, waist, and lower back (cortisol-driven visceral fat storage, the most common presentation in women over 35) that has appeared in the last few years without a clear dietary or lifestyle cause
Weight that has not budged in 6, 12, or 18 months (insulin resistance combined with elevated cortisol) despite eating well and moving regularly
Hunger an hour or two after a meal (ghrelin and leptin disruption from chronic cortisol) that should have been filling, particularly when you have eaten enough protein
Sugar and carbohydrate cravings between 2 and 4pm (cortisol-driven blood sugar drop) that you cannot reason your way out of
Waking between 2 and 4am with a racing mind (cortisol awakening response disruption from blood sugar instability) and struggling to get back to sleep for an hour or more
Tightness through the middle while the rest of the body still fits (insulin and cortisol depositing fat preferentially in the abdominal area where receptor density is highest)
Bloating and puffiness that worsens through the day (water retention from oestrogen dominance and inflammation rather than dietary sensitivity alone)
Fatigue that does not improve with sleep (functional hypothyroidism from impaired T4 to T3 conversion, often masked by "normal" thyroid bloods)
Loss of muscle definition in the arms, shoulders, and legs (sarcopenia from years of under-eating, cardio-only training, or chronic cortisol elevation)
Cold hands and feet, dry skin, thinning hair (low active thyroid hormone from poor T4 to T3 conversion in the liver)
Heavier or more painful periods alongside the weight gain (oestrogen dominance with poor liver clearance) where the cycle has changed at the same time as body composition
Increased abdominal weight in perimenopause (declining oestrogen reducing insulin sensitivity and shifting fat distribution from hips and thighs to belly)
Fluid retention in the fingers, ankles, and face (oestrogen, insulin, and cortisol all affecting fluid balance)
A sense that your metabolism has slowed in the past 2 to 5 years (combined insulin resistance, cortisol load, thyroid suppression, and muscle mass loss compounding)
Strong resistance to weight loss while on or after a GLP-1 medication (loss of muscle mass during medicated weight loss reducing the metabolic foundation needed to maintain results)
This is what I look at in clinic.
Insulin is the hormone your pancreas releases every time you eat. Its job is to clear glucose out of your bloodstream and move it into your cells, either to be used for energy or stored for later. Healthy insulin function is fast, efficient, and quiet. You eat, insulin rises, glucose moves into the cells, insulin drops back to baseline, and your body switches to burning stored fat for energy in the gap until the next meal.
Insulin resistance is what happens when this system has been overworked for years. After repeated exposure to high circulating insulin (from frequent carbohydrate-heavy meals, snacking through the day, chronic stress raising blood sugar through cortisol, poor sleep, or all of these stacked together), your cells become less responsive to insulin's signal. They stop opening the door as easily. Your pancreas compensates by producing more insulin to push the same amount of glucose into the cells, and the higher your circulating insulin sits, the harder it is for your body to access stored fat. Insulin is a fat-storing hormone. While it is elevated, your fat cells are locked, and no amount of dietary restriction or exercise will override that hormonal signal.
The most useful marker for this is fasting insulin, which most GPs do not run. A fasting glucose can sit within range for years while fasting insulin climbs, because the pancreas is working harder and harder to keep glucose normal. By the time fasting glucose looks abnormal on a standard panel, insulin resistance has often been present for 5 to 10 years. From a functional perspective, fasting insulin around 4 indicates good cell sensitivity and minimal pancreatic strain. Fasting insulin around 7 or 8 is the beginning of insulin resistance, the metabolic strain is already underway (fat storage increasing, inflammation building, hormones being affected), and most GPs would call this number normal. Fasting insulin above 10 is insulin resistance, and the body is in active fat storage with impaired fat burning.
Insulin resistance gets worse with age in women because oestrogen plays a protective role in cell sensitivity. As oestrogen fluctuates and declines through perimenopause, your cells become less responsive to insulin, your pancreas produces more of it, and the fat storage pattern accelerates around the belly, the upper body, and the upper arms.
This is one of the reasons women in their late 30s, 40s, and 50s start gaining weight in those specific areas for the first time despite no change in their diet or activity level. The hormonal environment has shifted, and the food and exercise that used to work no longer match the body running underneath them.
Insulin sensitivity is one of the fastest systems to respond to clinical change. Stable blood sugar, longer gaps between meals, lower carbohydrate intake, adequate protein, and resistance training all directly improve insulin signalling, often within weeks.
When cortisol is short-lived (acute stress, the threat resolves, cortisol drops back down), it is a useful, even essential hormone. The problem for most women in their 30s, 40s, and 50s is that the threat never fully resolves. The job pressure does not lift. The caregiving load is constant. The sleep is interrupted. The blood sugar is unstable. Cortisol stays elevated for weeks, months, and years, and the body adapts to that elevation as the new baseline.
Chronically elevated cortisol does several things to body composition at once. The first is direct fat storage in the abdominal area. Visceral fat cells (the fat stored deep around your organs) have approximately four times more cortisol receptors than the subcutaneous fat cells stored under your skin on your arms, legs, and hips. This means cortisol preferentially deposits fat around the midsection. The thicker waist, the belly that pinches over the waistband of your jeans, the weight that sits in front and around the sides while the rest of your body looks much like it always has, is a cortisol-driven fat distribution pattern. Calorie restriction and abdominal exercise will not override this hormonal signal while cortisol stays elevated.
The second is appetite hormone disruption. Cortisol increases ghrelin (the hormone that signals hunger to your brain) and reduces leptin (the hormone that signals fullness). The result is that you are hungrier more often, harder to satisfy, and reaching specifically for sugar, refined carbohydrates, and fatty foods because your body is asking for quick fuel for a perceived emergency. The 3pm pantry visit, the evening cravings, the feeling of being hungry an hour after a meal that should have been filling, are hormonal responses to cortisol-disrupted appetite signalling.
The third is the cortisol-insulin loop. Cortisol triggers your liver to release stored glucose into the bloodstream (a process called gluconeogenesis), which raises blood sugar even when you have not eaten. That blood sugar rise triggers insulin to clear it, and now you have both cortisol and insulin elevated at the same time. Both are fat-storing hormones. Both promote inflammation. Both block fat burning. Your body is locked into active storage mode while simultaneously losing the ability to access what is already stored.
I see this loop in clinic constantly in women who have done everything right. The food is good. The training is happening. The weight will not move because the hormonal environment created by chronic stress is promoting fat storage from every direction at once.
The thyroid problem I see most in women presenting with stubborn weight gain is not low thyroid hormone production. The thyroid gland is often working fine. The problem is that T4 is not being converted into T3 efficiently, which means even though the bloods look adequate, the active hormone your cells actually need is sitting low. The standard thyroid panel from a GP usually only checks TSH and T4. If both are within range, you are told your thyroid is fine, even when every symptom you have (the fatigue, the cold hands, the weight gain, the brain fog, the dry skin, the thinning hair, the constipation, the slow recovery from exercise) is pointing toward functional hypothyroidism.
The conversion of T4 into T3 is impaired by chronic cortisol, elevated insulin, liver congestion, gut dysbiosis, and nutrient deficiencies (selenium, zinc, iron, iodine, and tyrosine).
Almost every woman presenting with stubborn weight gain has at least three of those running at the same time. Cortisol also increases the conversion of T4 into reverse T3, which is an inactive form of thyroid hormone that competes with active T3 at the receptor sites on your cells. So even the small amount of T3 that is being produced has fewer receptor sites available to bind to, and the metabolic signal is weakened from both directions.
The functional thyroid panel I run in clinic includes TSH, free T4, free T3, reverse T3, and TPO and thyroglobulin antibodies. The pattern I find most often in women with stubborn weight gain is TSH normal, T4 normal, T3 low, and reverse T3 elevated. The thyroid gland is producing the hormone. The conversion is where the breakdown is occurring, and the cause is almost always sitting in the liver, the gut, the cortisol load, or the nutrient status, which is why the work is rarely just thyroid.
Resting metabolic rate is the number of calories your body burns while doing nothing, and active T3 is one of the primary regulators of that number. When T3 sits low, resting metabolic rate drops, and your body becomes more efficient at storing what comes in and slower at burning what is already there. This is one of the reasons women with functional hypothyroidism describe feeling like their metabolism has slowed even when their food and exercise have not changed.
The number on the bloods is not telling the full story.
Oestrogen and progesterone work in a paired rhythm across your cycle, and that rhythm changes through your 30s, 40s, and into menopause. Progesterone is the calming hormone. It supports deep sleep, stabilises mood, regulates the second half of your cycle, and balances the effects of oestrogen. Progesterone starts to decline earlier than oestrogen, often in the late 30s, which is why many women notice their first signs of hormonal change as worsening sleep, increased anxiety, shorter luteal phases, and more pronounced PMS years before they would consider themselves perimenopausal. Lower progesterone also means oestrogen sits relatively higher even when absolute levels have not increased, which is the pattern called oestrogen dominance.
Oestrogen dominance contributes to weight gain in several specific ways. The first is fluid retention, particularly through the cycle and especially in the lead-up to a period. The bloating, the puffiness in the fingers and the face, the 1 to 2kg the scale picks up that disappears a few days into the next cycle, are oestrogen-driven fluid shifts rather than fat gain. The second is fat distribution. Oestrogen promotes fat storage in the hips, the thighs, the buttocks, and the breasts. Higher relative oestrogen often shows up as weight gain in those areas alongside the abdominal weight from the cortisol and insulin pattern.
The third is liver and gut load. Used oestrogen has to be cleared by the liver and excreted via the bowel, and when liver function is congested or the gut microbiome is out of balance, oestrogen recirculates instead of being eliminated. This worsens oestrogen dominance regardless of how much oestrogen the ovaries are actually producing. Insulin compounds this further, because elevated insulin promotes the conversion of testosterone and DHEA into oestrogen in fat tissue, which means the more insulin resistance is present, the more oestrogen is being produced in the body's own fat cells. The cycle reinforces itself.
Through perimenopause, oestrogen does not decline smoothly. It swings, and that erratic pattern compounds the weight picture by repeatedly disrupting insulin sensitivity. As oestrogen levels drop further into menopause, the protective effect oestrogen has on insulin sensitivity is lost, fat redistribution shifts further toward the abdomen, and the progesterone decline that started in the late 30s becomes more significant.
The clinical pattern I see most often in women in this stage is a combined picture: low progesterone, fluctuating or relatively elevated oestrogen, declining DHEA from chronic stress, impaired thyroid conversion, and insulin resistance, all running at once. The weight gain is the visible end of a hormonal sequence that has been compounding for 5 to 10 years.
Your gut is doing more for your weight than you can see from the outside. The microbiome (the trillions of bacteria living in your digestive tract) influences how much glucose your body extracts from a given meal, how much insulin is released in response, how efficiently used hormones are cleared, how much inflammation is circulating, and how much water your body holds. None of this shows up on a scale as a separate number, but every part of it shapes the number you see.
The first connection is microbial composition. Some bacterial populations are more efficient at extracting energy from food than others. Two women eating the same meal can absorb different amounts of glucose depending on the makeup of their microbiome, which means the same calorie intake can produce a different metabolic outcome. Women with low microbial diversity (often from years of antibiotics, restrictive eating, low-fibre diets, or chronic stress) tend to have higher inflammatory markers, less efficient blood sugar regulation, and stronger cravings for sugar and refined carbohydrates because the bacteria themselves signal for the food that keeps them dominant.
The second connection is short-chain fatty acid production, particularly butyrate. Beneficial gut bacteria produce butyrate when they ferment fibre from vegetables, legumes, nuts, and seeds. Butyrate strengthens the gut wall (think of it like re-grouting tiles in a bathroom, sealing the gaps so nothing leaks through into the bloodstream), reduces intestinal inflammation, and improves insulin sensitivity directly at the cellular level. When the microbiome is depleted or out of balance, butyrate production drops, the gut wall becomes more permeable, inflammatory compounds cross into the bloodstream, and the liver has to manage that inflammatory load alongside everything else it is processing.
The third connection is the estrobolome, a specific group of gut bacteria responsible for clearing used oestrogen from the body. When the estrobolome is dysbiotic, packaged oestrogen is reactivated in the gut and recirculated instead of being eliminated, which contributes to oestrogen dominance, fluid retention, and the hormonal weight pattern that often shows up alongside heavier or more painful periods.
The fourth connection is inflammation and water retention. A compromised gut wall allows bacterial fragments and partially digested food particles into the bloodstream, which the immune system responds to with low-grade systemic inflammation. Inflammation increases insulin resistance (worsening the storage signal), promotes water retention (showing up as puffiness in the face, fingers, and ankles), and slows recovery from exercise (making it harder to build the muscle that supports metabolic rate). The bloating that worsens through the day, the puffiness in the morning, the sense that your body is holding more fluid than it should, are often gut-driven rather than dietary in origin.
I see this in clinic regularly, and the gut work is rarely optional in stubborn weight cases. Until the microbiome is supported, butyrate production is restored, the gut wall is repaired, and the estrobolome is rebalanced, the metabolic and hormonal work has a ceiling.
Muscle is the engine of your metabolism. Every kilogram of muscle on your body burns calories at rest, supports insulin sensitivity, regulates blood sugar, produces compounds that reduce inflammation, and makes your body a more efficient fat-burner around the clock. The more muscle you carry, the higher your resting metabolic rate, the better your body handles carbohydrates, and the easier it is to maintain a healthy body composition through hormonal change. Muscle is the single most underrated factor in long-term weight management for women, and it is the one that most diet advice ignores.
The problem is that the way most women have been told to lose weight (eat less, do more cardio, repeat for years) actively destroys muscle. Sustained calorie restriction signals your body to break down muscle tissue for energy, particularly when protein intake is too low to preserve it. Cardio without resistance training does very little to build or protect muscle. And every cycle of restrictive dieting followed by regain replaces some of the lost weight as fat instead of muscle, which is why women who have spent 10 or 15 years yo-yoing often find themselves with a smaller frame, higher body fat percentage, and slower metabolism than when they started.
Cortisol compounds this directly. Chronic cortisol breaks down muscle tissue (a process called muscle catabolism) to release amino acids the body uses for glucose production through the liver. So women who are under chronic stress, eating low-protein meals, restricting calories, and doing high-intensity cardio without recovery are losing muscle from multiple directions at once. The metabolic rate drops year on year, and the same eating habits that maintained weight 5 years ago now produce slow weight gain, because the body running underneath is no longer the same body.
GLP-1 medications add a specific layer to this picture.
Studies on rapid weight loss through GLP-1 use have shown significant muscle mass loss alongside the fat loss, often 25 to 40% of the total weight lost being lean tissue. For women already low in muscle from years of dieting, this can mean coming off the medication with a much lower metabolic rate than they started with, which is one of the reasons regain after stopping a GLP-1 is so common and so fast. The work of supporting muscle mass before, during, and after GLP-1 use is one of the most important pieces of long-term outcomes, and it is the part most prescribing pathways do not address.
Building and maintaining muscle requires adequate protein, resistance training, and enough food to support the work. The instinct most women have when the scale will not move is to eat less. The clinical reality for women with stubborn weight gain is that under-eating is often part of why the weight will not move, and restoring metabolic rate requires feeding the body enough to rebuild the tissue that does the burning.
I see this in clinic constantly. Women who are eating 1200 calories, doing daily cardio, and gaining weight, because the muscle has gone, the cortisol is high, the thyroid is suppressed, and the body has adapted to survive on less. The intervention is rarely more restriction. The intervention is rebuilding the metabolic foundation.
Food as Medicine
Nutrition is the foundation of your weight loss plan, and thanks to my Graduate Certificate in Culinary Nutrition Science alongside my clinical qualifications, your food plan is delicious, practical, and designed around your life.
It is built around what your body needs through your specific clinical presentation, not a generic template pulled off the internet.
I look at your symptoms, blood sugar response, inflammation levels, nutritional gaps, and the specific foods that support the systems under most pressure in stubborn weight gain: insulin and blood sugar, hormones, thyroid, gut, and nervous system.
You also get a custom recipe book matched to your plan, so you are never staring at a protocol wondering what to cook on a Tuesday night.
The goal is a way of eating you can sustain for the long term, that still lets you enjoy a margarita or pizza on a Friday night.
Supplements and Herbal Medicine
Supplements and herbal medicine are two of the most powerful clinical tools I use in stubborn weight gain, and you get access to practitioner-only prescriptions built specifically for your body.
These are the highest-quality clinical formulations available in Australia, with therapeutic doses and activated forms that absorb and work in the body.
A different tier entirely to anything on a chemist shelf.
Your protocol is matched to your stubborn weight gain symptoms, test results, and the systems we are working on, whether that is insulin sensitivity, hormonal balance, thyroid function, gut health, or appetite regulation.
Herbal medicine is one of the oldest and most evidence-backed forms of medicine in existence, and when prescribed correctly for stubborn weight gain, it often produces results faster than women expect.
I adjust your prescription across the package as your body responds, so you are always taking what is clinically useful and nothing you do not need.
Nervous System Regulation
Your nervous system regulates almost every other system in the body, and when it is held in a chronic stress state during stubborn weight gain, cortisol stays elevated, insulin resistance worsens, fat storage is promoted around the midsection, and metabolic flexibility is impaired.
Chronic stress is one of the most significant contributors to stubborn weight gain symptoms, and it is the part most treatment plans overlook.
Your plan includes clear, practical strategies that fit into a busy life: vagus nerve support, short breathwork practices, sleep support including circadian rhythm work and clinical strategies if your sleep is disrupted, stress regulation built around your capacity, and movement that supports your system rather than adding to it.
Most women notice improvement within the first few weeks.
Advanced Testing & Integrative Pathology Review
You have access to advanced functional testing when it is clinically useful for your case and your budget.
This includes fasting insulin and HbA1c for insulin resistance, DUTCH Complete for cortisol and hormonal contributors, full thyroid panel including reverse T3, and GI-MAP for gut-weight connection, alongside DNA and nutrigenomic testing, organic acids, and food sensitivity testing when indicated.
I only recommend testing that is going to meaningfully inform your treatment direction.
I also review your pathology through an integrative lens using functional optimal ranges, which are tighter than standard population reference ranges.
A result can sit inside the "normal" range and still be contributing to your stubborn weight gain symptoms, and this is often where the clinical answers are found.
80/20 Always
You can have the wine. You can have the margarita on a Friday. You can have the pasta, the cheese platter, and the slice of birthday cake.
Restriction, perfectionism, and constant dieting work against your hormones, your nervous system, and your long-term consistency, which is the last thing your body needs in stubborn weight gain.
They also compromise your relationship with food, which is one of the most difficult things to repair in women in this stage of life.
Your plan is built to be sustainable for life, not for 30 days, so you can navigate stubborn weight gain and enjoy your life at the same time.
The goal is a way of eating and living that fits into a full social life and continues to support your clinical outcomes.
Realistic & Built Around Your Life
Your plan is designed around how busy you are, your schedule, who you cook for, how much time you have, and the capacity you are carrying in this stage of life.
Simple, sequenced, and achievable.
I will not hand you a protocol that requires hours in the kitchen, a fridge full of obscure ingredients, or a morning routine longer than the average commute.
No plan produces results if it does not fit your life, which is why most generic programs fail women in stubborn weight gain.
Yours is built to fit yours, step by step, in the clinical order your body needs through your specific presentation.
Naturopath In Your Pocket
Between consults, you have direct messaging access to me for questions about your protocol, new symptoms you want to flag, situations that change the plan, or progress you want to share.
You are not waiting for weeks with a clinical question until your next appointment, and you are not working through the complex parts on your own.
Stubborn weight gain symptoms shift month to month, and having clinical support between sessions makes a significant difference to outcomes across a multi-month protocol.
Most women tell me this is one of the most valuable parts of working together.
Mindset & Your Relationship With Food
Most women I see in stubborn weight gain already have a bit of an idea of what they need to do.
They have read the books, followed the accounts, bought the supplements, and started the protocols. Some have been on a GLP-1 medication and are working out what comes next. Some are considering one. Some are doing the work alongside it.
What keeps them stuck is what is underneath: all-or-nothing thinking, self-sabotage when progress begins, food guilt, body image concerns, and years of yo-yo dieting that now feel amplified in this stage of life.
This thinking consistently undermines every protocol a woman has tried before.
Your plan includes work on these patterns as part of your clinical care, because a treatment plan does not produce lasting results if the thinking underneath it is pulling in the opposite direction.
This is part of your care, not an extra.
Yes. A naturopath and nutritionist can be one of the most useful practitioners to see in stubborn weight gain because the approach is root-cause and systems-based. I look at insulin, cortisol, hormones, thyroid, gut, liver function, and nutrition together rather than treating one symptom at a time. For Australian women who feel dismissed by conventional care, or who want support alongside GLP-1 medications, hormone therapy, or thyroid medication, it is often the part they have been missing.
A GP is usually your first stop for diagnosis, medication, and standard pathology, which is valuable clinical care. A naturopath and nutritionist takes a root-cause, holistic approach to your health, because what happens in one system of the body affects every other system. Your hormones, gut, thyroid, nervous system, and metabolism all communicate with each other, and treating them in isolation rarely gives lasting results.In clinic, I use functional testing (DUTCH, GI-MAP, fasting insulin, full thyroid panels), practitioner-only supplements, custom herbal medicine, and personalised nutrition to support all of these systems together.This is a comprehensive, intensive, and hands-on approach. You get a personalised clinical plan, ongoing support between consults, and the time to work through your stubborn weight gain symptoms with the depth they need.The two approaches are complementary, not either-or. I work with many women who are on GLP-1 medications, hormone therapy, or thyroid medication through their GP and working with me on the gut, nutrition, nervous system, muscle mass, and lifestyle foundations that medication does not replace.
Energy and digestion usually start to improve in the first 2 to 4 weeks, and most women start to see shifts in their weight in the first month. Mood, skin, and early symptom relief follow between 4 and 8 weeks. Consistent weight shifts and metabolic flexibility come across the 3 to 6 month mark. Every woman is different, but most describe significant improvement within the first few months of working together.
Yes. I work with women who are on, considering, or recently off a weight loss medication, including GLP-1 medications like Ozempic, Mounjaro, and Wegovy.
The work I do alongside these medications is the foundational work the medication itself does not do. That includes the nutrition that supports muscle preservation during weight loss (which is one of the biggest issues with rapid GLP-1 weight loss), the protein and resistance training framework that protects metabolic rate, the gut and liver work that supports hormonal clearance, and the mindset and food relationship piece that determines what happens when you stop the medication.
I do not prescribe or deprescribe any medication. That conversation belongs with your GP or specialist. What I do is support the foundations (gut, nutrition, nervous system, muscle mass, mindset, lifestyle) so that whatever you decide about the medication, your body has the metabolic and hormonal foundation to maintain results long-term.
Tests vary depending on each person, and recommendations are based on your history and symptoms. Not every woman needs every test.
Commonly, I will run fasting insulin and HbA1c for insulin resistance, DUTCH Complete for cortisol and hormonal contributors, full thyroid panel including reverse T3, and GI-MAP for the gut-weight connection.
Where it is clinically useful and fits your budget, I also use DNA and nutrigenomic testing, organic acids, and food sensitivity testing.
No. Naturopaths and nutritionists in Australia do not require a GP referral. You can book directly.
Yes. All my consultations run online via telehealth, which means I work with women in every state and territory. Most of my clients are in Sydney, Melbourne, Brisbane, Perth, Adelaide and regional NSW, but geography is not a barrier.
The initial consultation is 90 minutes and includes a personalised plan you leave with. Follow-up consultations run 30 to 60 minutes depending on what is needed. Testing is additional and quoted up front so there are no surprises. Full pricing is on the Consultations page. Private health rebates may apply depending on your fund.
Yes. I can organise pathology and functional testing directly through the private labs I use in clinic, including full functional blood panels, DUTCH Complete, DUTCH Cycle Mapping, GI-MAP, DNA and nutrigenomic testing, and other specialised tests when indicated.
These are out of pocket because they are not covered by Medicare. Some standard blood tests are covered by Medicare when ordered through a GP, so for those panels I will write you a clear list of exactly what to ask your GP for, which keeps the cost down.
Functional tests like DUTCH and GI-MAP are not available through Medicare or the public system, so these are always out of pocket. I will give you the cost up front before we commit to any testing, and I will only ever recommend testing that is going to meaningfully inform your clinical plan.
Not forever. Not all at once. I work on the 80/20 rule personally and professionally. There will be periods where reducing certain things speeds the process up (particularly in the early weeks of rebalancing blood sugar or the gut), but the plan is always built for sustainability.
You can have the glass of wine. You can have the margarita. Restriction is not the strategy.

Bachelor of Health Sciences (Naturopathy)
Master of Advanced Naturopathic Medicine
Graduate Certificate in Culinary Nutrition Science
A5M certification in anti-ageing medicine
8+ years of clinical practice
1,000+ women worked with
Australian Nutrition Ambassador for MyFitnessPal
© Michaela Sparrow 2026